There's been another one of those genetic discoveries that could be far-reaching in its implications, namely, Japanese scientists have found a link between the emerging field of epigenetics, and aging (and thanks to Mr. J.H. for sharing this article):
The key hereare these two paragraphs:
The Tsukuba team in particular has performed some compelling research that has led them to propose that age-associated mitochondrial defects are not controlled by the accumulation of mutations in the mitochondrial DNA but by another form of genetic regulation. The research, published this month in the prestigious journal Nature’s ‘Scientific Reports’, looked at the function of the mitochondria in human fibroblast cell lines derived from young people (ranging in age from a fetus to a 12 year old) and elderly people (ranging in age from 80-97 years old). The researchers compared the mitochondrial respiration and the amount of DNA damage in the mitochondria of the two groups, expecting respiration to be reduced and DNA damage to be increased in the cells from the elderly group. While the elderly group had reduced respiration, in accordance with the current theory, there was, however, no difference in the amount of DNA damage between the elderly and young groups of cells. This led the researchers to propose that another form of genetic regulation, epigenetic regulation, may be responsible for the age-associated effects seen in the mitochondria.
Epigenetic regulation refers to changes, such as the addition of chemical structures or proteins, which alter the physical structure of the DNA, resulting in genes turning on or off. Unlike mutations, these changes do not affect the DNA sequence itself. If this theory is correct, then genetically reprogramming the cells to an embryonic stem cell–like state would remove any epigenetic changes associated with the mitochondrial DNA. In order to test this theory, the researchers reprogrammed human fibroblast cell lines derived from young and elderly people to an embryonic stem cell-like state. These cells were then turned back into fibroblasts and their mitochondrial respiratory function examined. Incredibly, the age-associated defects had been reversed - all of the fibroblasts had respiration rates comparable to those of the fetal fibroblast cell line, irrespective of whether they were derived from young or elderly people. This indicates that the aging process in the mitochondrion is controlled by epigenetic regulation, not by mutations.
The field of epigenetics is new, but growing quickly, and opens up rather obvious speculative implications, among them, the role of nutrition, and, by implication, the possible epigenetic influence of GMOs vs non-GMO foods, as well as the influence of pesticides and other environmental influences on DNA and cellular health.
But I want to indulge in a bit of different speculation here, one that I suspect will shortly emerge as epigentic centers of study and concern, and that's the electromagnetic effects on DNA. Retired US Army Lt. Colonel Tom Bearden, years ago, pubished a book titled Gravitobiology, detailing electromagnetic inductions, and cures, of diseases that was undertaken in extremely classified research inside the Soviet Union, a glimpse of which appeared in Soviet scientific journals. The essence of this research consisted in the fact that every healthy cell has a kind of electromagnetic signature, whereas diseased cells had their own signatures, unique to whatever disease - cancer among them - they were suffering. Soviet scientists allegedly discovered that by producing an electromagnetic field with healthy(or diseased) signatures on a target area, cells in that area would revert to whatever characteristic was being beamed. In effect what the Soviets were doing wa modulated the electromagnetic signature onto a carrier wave, then irradiating the tissue with it. In similar fashion, such studies were conducted (and quickly repressed) by Priore in France, and, of course, in very different fashion, by Dr. Royal Raymond Rife in this country.
So what's the high octane speculation here(besides Col. Bearden's, which is not really "high octane" but a rather well argued case)? The speculation is more by way of prediction: I suspect we shall see, gradually at first, then more and more of a steady stream, of studies of epigenetic effects of electromagnetic fields, both in the form of deliberate targeting (as in Bearden's examples in his book), and also in the form of electromagnetic pollution studies. That in turn will leads to renewed calls for "electromagnetic pollution regulation," and the rest of the scenario can be guessed at. The real crunch will come - possibly - if it can be demonstrated that specific genetic mutations can be induced electromagnetically, and then inherited, in a kind of electromagnetic epigenetic genetic engineering.
See you on the flip side...